C 022: THE GUANINE-BASED PURINERGIC SYSTEM AS A NEW TARGET FOR NEUROPROTECTION AGAINST GLUTAMATERGIC EXCYTOTOXICITY

Excerpt:


J Pharm Pharmacogn Res 2(Suppl. 1): S65, 2014 Special supplement with the abstract book of LATINFARMA 2013 Conference C 022: THE GUANINE-BASED PURINERGIC SYSTEM AS A NEW TARGET FOR NEUROPROTECTION AGAINST GLUTAMATERGIC EXCYTOTOXICITY Souza DO. Departamento de Bioquímica, PPG em Bioquímica e PPG em Educação em Ciências, ICBS, UFRGS. Rua Ramiro Barcelos, 2600 – Anexo. … Continue reading C 022: THE GUANINE-BASED PURINERGIC SYSTEM AS A NEW TARGET FOR NEUROPROTECTION AGAINST GLUTAMATERGIC EXCYTOTOXICITY

J Pharm Pharmacogn Res 2(Suppl. 1): S65, 2014

Special supplement with the abstract book of LATINFARMA 2013

Conference

C 022: THE GUANINE-BASED PURINERGIC SYSTEM AS A NEW TARGET FOR NEUROPROTECTION AGAINST GLUTAMATERGIC EXCYTOTOXICITY

Souza DO.

Departamento de Bioquímica, PPG em Bioquímica e PPG em Educação em Ciências, ICBS, UFRGS. Rua Ramiro Barcelos, 2600 – Anexo. CEP: 90035-003, Porto alegre, RS, Brazil.
Abstract

Glutamate is the main excitatory neurotransmitter in mammalian CNS, essential for brain activities, as those involved in development, ageing, memory, and adaptation to the environment. However, hyper activation of the glutamatergic system may be potentially neurotoxic, involved in the pathogenesis of various acute and chronic brain injuries. The main process responsible by maintaining the extracellular glutamate levels below toxic levels, thus favoring the physiological glutamatergic tonus, is the glutamate uptake exerted by transporters located in neural cell membranes, mainly in astrocytes. Our group has given strong evidence that the guanine-based purinergic system is effectively neuroprotective against glutamate toxicity, in acute and chronic animal models, both in vitro and in vivo studies. Our results indicate that nucleoside guanosine (Guo) exerts neuroprotection. In vivo Guo i.c.v., i.p. or orally administered in rodents protects against brain injury caused by overstimulation of the glutamatergic system. In vitro studies, Guo protects cell death in brain slices caused by in vitro ischemia. Searching for mechanisms implicated in this neuroprotection, we demonstrated that: i) Guo stimulates the astrocytic glutamate uptake (in astrocyte cultures and brain slices), the main process involved in endogenous neuronal protection; ii) brain induced-injuries decrease glutamate uptake by brain slices and this decrease is reversed by Guo only when it acts as anticonvulsant; iii) brain oxidative stress caused by glutamate toxicity in experimental brain injury models is reversed by Guo administration. Thus we propose that the stimulatory effect on glutamate uptake and/or its antioxidant activity are involved in the neuroprotective actions of Guo. These results encourage further studies aiming at the therapeutic use in humans of Guo in acute (hypoxia, ischemia, brain traumatism) and chronic (neurodegenerative diseases) brain injuries involving glutamate excytotoxicity.

Toxicity evaluation of Ruta angustifolia leaves ethanolic extract
J. Pharm. Pharmacogn. Res., vol. 11, no. 3, pp. 437-447, May-June 2023. DOI: https://doi.org/10.56499/jppres23.1609_11.3.437 Original Article Acute and repeated dose 28-day oral toxicity of Ruta angustifolia Pers. leaves ethanolic extract in Wistar rats [Toxicidad oral aguda y por dosis repetidas durante 28 días del extracto etanólico de hojas de Ruta angustifolia Pers. en ratas Wistar] Tutik … Continue reading Toxicity evaluation of Ruta angustifolia leaves ethanolic extract
Adolescents in Indonesia and COVID-19 pandemic
J. Pharm. Pharmacogn. Res., vol. 11, no. 3, pp. 426-436, May-June 2023. DOI: https://doi.org/10.56499/jppres22.1560_11.3.426 Original Article Perception, mental health, and social media exposure on adolescents in Indonesia during COVID-19 pandemic [Percepción, salud mental y exposición a los medios sociales en adolescentes de Indonesia durante la pandemia de COVID-19] Efa Nugroho1*, Alfiana Ainun Nisa1, Widya Hary Cahyati1, … Continue reading Adolescents in Indonesia and COVID-19 pandemic
Decaffeinated green tea and coffee extracts and metabolic syndrome
J. Pharm. Pharmacogn. Res., vol. 11, no. 3, pp. 414-425, May-June 2023. DOI: https://doi.org/10.56499/jppres23.1593_11.3.414 Original Article Decaffeinated green tea and green coffee extracts as metformin’s add-on enhance metabolic syndrome risk factors and improve the cardiac insulin-gene-related pathway [Extractos de té verde y café verde descafeinados como complemento de la metformina mejoran los factores de riesgo del … Continue reading Decaffeinated green tea and coffee extracts and metabolic syndrome

© 2013-2020 by the authors; licensee JPPRes, Antofagasta, Chile. This journal is an open-access journal distributed under the terms and conditions of the Creative Commons Attribution license-Non Commercial 4.0 international. The content on this site is intended for health professionals. If you are not a health professional, please talk to your doctor about any doubts or concerns regarding your health

Made with ♥ by AVAGAX Studio